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Fig. 5 | BMC Developmental Biology

Fig. 5

From: Neural defects caused by total and Wnt1-Cre mediated ablation of p120ctn in mice

Fig. 5

No prominent cell proliferation changes in p120fl/fl;Wnt1Cre mutant mice. Sections through the hindbrain region of 12–13 somite embryos are shown. a, b Expression of p120ctn was prominent in the neural folds of a control embryo (a), but ablated in the neural folds of a mutant embryo (b, arrows). c, d Immunodetection of phosphorylated Histone 3 (pH 3) showed similar activity in control and mutant embryos. Scale bar: 20 μm

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